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The current standard of care for the treatment of HF is the administration of a diuretic, a drug that inhibits sodium reabsorption in the kidney tubules. Despite diuretic therapy, many patients with HF are chronically volume overloaded and exhibit fluid retention. The resultant increase in blood volume causes a further drop in cardiac output and exacerbates the symptoms of HF. In the advanced stages of HF, fluid retention leads to ascites, peripheral edema and pulmonary edema.
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